Acute glomerulonephritis after streptococcal infection:
And iga kidney disease is also prone to young men, in the upper respiratory tract infection (or acute tonsillitis) after hematuria, may have proteinuria, edema and high blood pressure, and even renal damage. The difference is that iga nephropathy in patients with upper respiratory tract infection after a short interval (1-3 days) that hematuria, some patients with elevated serum iga levels. And acute nephritis more than 2 weeks after the infection of streptococcal acute nephritis syndrome clinical symptoms, serum c3 decline, iga level can help identify the normal.
Non iga mesangial proliferative glomerulonephritis:
Non iga mesangial proliferative glomerulonephritis in China. About 1/3 of the patients showed simple hematuria. In clinical practice with iga kidney disease is difficult to identify. To be identified by renal biopsy immunopathology.
Thin basement membrane nephropathy:
Thin basement membrane nephropathy mainly clinical manifestations of repeated hematuria, about 1/2 cases of family history. Clinical manifestations of benign process. Urine pf4 levels can help with iga kidney disease identification. Must be checked by renal biopsy and iga kidney disease identification.
Allergic purpura nephritis:
Patients can be expressed as microscopic hematuria and even gross hematuria. Renal biopsy may have the same extensive imala deposition as the primary iga nephropathy. However, patients with purpura kidney often have typical skin purpura, abdominal pain, joint pain performance.