Rheumatic nephritis dietary guidelines

Rheumatism is main diseases of the musculoskeletal system in the treatment of a medicine, in addition to rheumatism involving muscle, near the joint and soft tissue, such as systemic lupus erythematosus, systemic vasculitis often involving the kidneys, brain, heart and other vital organs, such as poor disease control, can cause severe organ dysfunction, loss of activity and even death.

Lancereaux nephritis (rheumatic interstitial nephritis), also known as rheumatoid nephritis

I. etiology

Acute rheumatic fever and acute glomerulonephritis are similar in etiology and pathogenesis is generally accepted, both have close relationship with streptococcal infection, and infection of allergy caused. There are two explanations: one is that caused by allergies; another is that by directly from rheumatism due. Can occur at the same time, but both the cases are very rare. It was confirmed 1%-40% patients with rheumatic heart disease in clinical and autopsy, especially adult kidney disease.

Two. Clinical manifestation

Mainly for proteinuria and hematuria, but found that the pathological changes of renal biopsy and acute nephritis was not the same. For interstitial nephritis.1959 Li Lianda reported 1 cases of acute rheumatic fever complicated with acute glomerulonephritis in children 14 years of age for rheumatic endocarditis, myocarditis, II degree atrioventricular blockade of acute glomerulonephritis.

Three. Diagnosis

In patients with rheumatic diseases, proteinuria, massive red blood cells (or even macroscopic hematuria) and a small number of tubular types should be considered in the absence of severe circulatory congestion and bacterial endocarditis

Four. Treatment

The main treatment should be anti infection, anti rheumatic treatment, early use of corticosteroids, but aspirin analgesics should not be used

Dietary guidelines for patients with rheumatic diseases

Eat less milk, milk and other goats'milk chocolate, peanut, millet, cheese, candy containing tyrosine, phenylalanine and tryptophan in food, produce medium prostaglandins, arthritis induced because of the leukotrienes, tyrosine kinase antibodies and anti IgE antibodies such as milk, prone to allergies caused by arthritis aggravation, recurrence or deterioration.

Eat less fat, high animal fat and high cholesterol foods, because of the resulting ketone, acid, four arachidonic acid metabolites and inflammatory mediators, can inhibit T lymphocyte function, easy to cause and aggravate the joint pain, swelling, destruction of bone demineralization and joint osteoporosis.

Eat less sweets, sugar because of its easy to cause allergy, can aggravate the development of synovitis, easy to cause the joint swelling and pain aggravation.

Drink less alcohol and coffee, tea and other beverages, and avoid passive smoking, which can exacerbate arthritis

What is rheumatism patient suitable to eat?

1, vegetables and fruits

Bitter gourd, bitter herbs, purslane, gourd and other heat effect, can alleviate the local fever;

Coix seed, bean curd, celery, lentils and so on, have the effect of clearing away heat and toxic substances, and it is good for relieving local pain;

Letinous edodes, black fungus and so on, help to improve human immunity, to alleviate local redness and swelling, heat pain and other symptoms effective;

Yam has Qi, Yin, kidney, spleen and lung role, can improve rheumatoid rheumatism, thirst, fatigue, sweating and other symptoms;

Black beans have nourishing Yin, invigorating spleen, qufengchubi effect, suitable for rheumatoid arthritis, spasm of limbs with disease, coix seed, papaya with better effect;

Medlar can strong bones, cold and heat, nourishing essence and fresh, with a white chrysanthemum soaked tea, also can eat porridge and rice;

In addition, a variety of vegetables, fruits are rich in human body needed vitamins, trace elements and cellulose, to improve metabolism is also very useful

2, pine nuts have nourishing the liver and kidney, brain and spleen, strong bones and muscles, rheumatoid patients daily edible 3 ~ 5 grams; chestnuts have Zhuangyao kidney, invigorating the spleen and stomach function, kidney, waist and knees may be appropriate to eat.

3, meat

Patients with rheumatoid arthritis by pigs, cattle, sheep and other joint bone or vertebral bone soup consumption, especially for rheumatoid acute, subacute, chronic bone decalcification, osteoporosis and can regulate the compensation.

The expert reminds: rheumatism easily caused by concurrent nephritis, therefore, suffering from rheumatic diseases, must pay special attention to their own conditions, any medical examination, to avoid deterioration caused by nephritis and other related diseases.

Rheumatic diseases and some medications can lead to several manifestations of kidney disease:

1. membranous nephropathy

Patients with nephrotic syndrome, but many RA patients were non nephrotic syndrome range proteinuria, after discontinuation of almost all patients with proteinuria can be alleviated, but in the first 1 to 12 months, excluding protein will continue to be increased, proteinuria disappeared time required for an average of 9 ~ 12 months.

2. nephropathy caused by analgesics

In the use of aspirin and phenacetin in treatment of rheumatoid arthritis patients often have chronic interstitial nephritis with renal papillary necrosis.

3. focal glomerulonephritis

Hematuria is usually associated with or without mild proteinuria

4. the glomerular basement membrane is thin

In patients with rheumatoid arthritis and asymptomatic hematuria, the basement membrane is thinner, but thinner than in thin basement membrane nephropathy

5. secondary amyloidosis

In some patients, amyloid deposits are confined to the renal vasculature, causing hematuria with or without small amounts of proteinuria

6. rheumatoid vasculitis

Occurs in erosive arthritis and significant nodule formation in patients with rheumatoid factor titers usually increased significantly with hypocomplementemia. Occurs most significantly for skin and peripheral nerves, renal lesions can occur, renal biopsy showed necrosis of renal glomerular inflammation without immune complex deposition.